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Design and synthesis of 4th generation EGFR inhibitors against human triple (Del19/T790M/C797S) mutation

  • Jiyoung Jeon
  • , Sun Young Jang
  • , Eun Joo Kwak
  • , Sun Hoe Lee
  • , Joo Yun Byun
  • , Yu Yon Kim
  • , Young Gil Ahn
  • , Pargat Singh
  • , Kyeongwon Moon
  • , In Su Kim
  • Sungkyunkwan University
  • Hanmi Pharmaceutical Co., Ltd.

Research output: Contribution to journalArticlepeer-review

Abstract

Epidermal growth factor receptor (EGFR)-targeted therapy is used to treat EGFR mutation-induced non-small cell lung cancer (NSCLC). However, its efficacy does not last beyond a certain period due to the development of primary and secondary resistance. First and second-generation inhibitors (e.g., gefitinib, erlotinib, and afatinib) induce EGFR T790M mutations, while third-generation inhibitors (e.g., osimertinib) induce C797S as a major target resistance mutation. Therefore, the C797S mutation is being actively researched. In this study, we investigated the structure-activity relationship of several synthesized compounds as fourth-generation inhibitors against the C797S mutation. We identified a compound 13k that displayed nanomolar potency and high selectivity. Moreover, we used a triple mutant xenograft mouse model to evaluate the in vivo efficacy of 13k in inhibiting EGFR C797S, which demonstrated exceptional profiles and satisfactory EGFR C797S inhibition efficacy. Based on its excellent in vitro and in vivo profiles, compound 13k can be considered a promising candidate for treating EGFR C797S mutations.

Original languageEnglish
Article number115840
JournalEuropean Journal of Medicinal Chemistry
Volume261
DOIs
StatePublished - 5 Dec 2023

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

  1. SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well-being

Keywords

  • C797S
  • EGFR
  • Inhibitors
  • NSCLC
  • Triple mutation

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